Sunday, July 28, 2019

Diet after wisdom tooth extraction


The first day(s) after the extraction of a tooth, you might be able to sustain yourself only with liquids. Aim at non-sugary drinks such as unsweetened almond or soy milk, regular milk, plain yogurt thinned with water and homogenized well. 


Once you feel you can transition to soft food, here are a few ideas:

  • oatmeal with a spoonful of each: peanut butter, fruit preserves and mashed banana
  • cream of wheat with a spoonful of hummus, mashed sweet potato and diced boiled egg
  • corn meal with a spoonful of mashed avocado.
  • oatmeal with a thinly diced soft peach or apricot and sweetened with a mashed banana.
  • cooked egg whites with hummus or a mashed avocado
  • blended soups

    • cottage cheese
    • plain yogurt with a spoonful of fruit preserves or plain yogurt with a drop of vanilla essence, mashed banana and a sprinkle of flaxseed
    • mashed potatoes

      • tofu pieces prepared on the stove top with some salt

      • tuna fish with mayo
      • dessert: ice cream, blended soft fruit, microwaved apple slices, applesauce

        Thursday, July 18, 2019

        Carbon Dioxide Secretion And Weight Loss

        Here is a hypothesis paper that suggests that carbon dioxide excretion maybe linked to problems with weight loss.  Abstract:

        Weight regulation depends on the difference between weights of absorbed and metabolised carbon and excretion of the end products of metabolism, calories and carbon dioxide, which can be independently and variably excreted. Calories can be variably excreted as heat by vasoconstriction or vasodilation and carbon dioxide can be excreted variably as exhaled carbon dioxide (the major route of carbon excretion). Unless there are changes in ventilatory carbon excretion, 'metabolism,' 'genetic factors,' 'hormones' or 'exercise' do not provide complete explanatory mechanisms for weight changes, obesity and failure of diets. Low sensitivity of respiratory centres to carbon dioxide may cause overweight and dietary failures after initial weight loss.

        Individuals less sensitive to carbon dioxide, and thus with impaired respiratory activity, may have more trouble with obesity and weight loss, opening up new avenues of prevention and treatment.

        Monday, July 8, 2019

        Vitamin D Against Colon Cancer

        Roles of vitamin D in colon cancer prevention, abstract:

        Colorectal cancer (CRC) is the neoplasia that is most frequently associated with vitamin D deficiency in epidemiological and observational studies in terms of incidence and mortality. Many mechanistic studies show that the active vitamin D metabolite (1α,25-dihydroxyvitamin D3 or calcitriol) inhibits proliferation and promotes epithelial differentiation of human colon carcinoma cell lines that express vitamin D receptor (VDR) via the regulation of a high number of genes. A key action underlining this effect is the multilevel inhibition of the Wnt/β-catenin signaling pathway, whose abnormal activation in colon epithelial cells initiates and promotes CRC. Recently, our group has shown that calcitriol modulates gene expression and inhibits protumoral properties of patient-derived colon cancer-associated fibroblasts (CAFs). Accordingly, high VDR expression in tumor stromal fibroblasts is associated with longer survival of CRC patients. Moreover, many types of immune cells express VDR and are regulated by calcitriol, which probably contributes to its action against CRC. Given the role attributed to the intestinal microbiota in CRC and the finding that it is altered by vitamin D deficiency, an indirect antitumoral effect of calcitriol is also plausible at this level. In summary, calcitriol has an array of potential protective effects against CRC by acting on carcinoma cells, CAFs, immune cells and probably also the gut microbiota.

        Tuesday, July 2, 2019

        The first FI genius in America



        The vast majority of us are sick and tired of their jobs, bosses and colleagues.

        We all want out. We all want freedom. There is a Peter Pan in each one of us. We still want to play. 



        This is why blogs about financial freedom and financial independence (FI) are becoming more and more popular.

        But how many of us know the way out of drudgery?

        Have you realized that the way out has been carefully obfuscated, made confusing and complicated?

        I am reading a great book that confirms my notion that there is a scheme. 


        ... Deliberately or not, the majority of the population is always kept on the edge of financial insecurity, if not poverty. They are not given the power of knowledge. Financial knowledge.

        The ONLY reason is to keep the majority of us working.

         

        The book I am reading is The Americanization of Benjamin Franklin by Gordon S. Wood. Here is a quote from this book that triggered today’s blog post:

        “…everyone but an idiot knows that the lower classes must be kept poor, or they will never be industrious.” 

          
        Arthur Young, 1771

        Well, today some people would be more industrious when un-tethered. But how to achieve the financial freedom? Unless you were a financial major in college, you may never get a few important lessons in finances and mostly, in personal finance. 


        Schools skip on the most critical lessons:

        1. Life is unfair

        2. Your health is your first priority

        3. How to master your personal finances

        I have previously reflected on the issues with the American educational system (here and here).  I have published on the future of America and its children.

        We need to learn the critical lessons on our own, if we are smart and curious enough. The life of Benjamin Franklin is one good lesson that we should not skip on.

        Let's find out how Franklin achieved his own financial freedom.

        He did so at a relatively young age.  In 1784, at age of 42, Franklin retired from work and took "the path of leisure". He acquired a few slaves and moved to a better, bigger house. 

        The leisure for this man, however, was education, experimentation, and Enlightenment. He learned foreign languages: Latin,French, Italian, Spanish, and German. He became a diplomat, statesman, civil activist, politician, scientist, and inventor.

        And yet, before all these achievements, he made it financially.


        How?

        He was a hustler.

        Franklin was a tradesman, printer, editor, writer, manager, and shop owner selling books and stationery, soap and cheese. 

        He took on any new opportunity to earn money: he became a clerk to the Pennsylvania Assembly and secured for himself the printing business of the organization. 

        Later, he became the public printer for New Jersey, Delaware, Maryland. 

        He started newspapers and magazines. 

        He wrote a famous almanac. 

        Served as a postmaster of Philadelphia and this helped his newspaper business.

        Partnered with many printers outside Philadelphia: by 1743, Franklin owned three printing firms in different colonies and was in partnership with many others. 

        Established many paper mills and became one of the largest paper dealer in the English-speaking world. 

        He bought rental properties.

        Became a creditor for loans small and big.

        Participated in land speculation.


        As a result, Franklin's income reached around £2,000 pounds a year.  If £100 in 1784 is equivalent in purchasing power to about £14,755.55, then today, in 2019, Franklin’s annual income would be £295,111 or $372,735.52.

        The financial independence achieved by Franklin allowed him to dedicate his talent and capabilities to creative and productive projects, with which he secured his recognition for centuries to come.

        Monday, July 1, 2019

        Lessons From Mice And Flies

        On Wnt signaling, abstract:

        Adult stem cells play critical roles in the basal maintenance of tissue integrity, also known as homeostasis, and in tissue regeneration following damage. The highly conserved Wnt signalling pathway is a key regulator of stem cell fate. In the gastrointestinal tract, Wnt signalling activation drives homeostasis and damage-induced repair. Additionally, deregulated Wnt signalling is a common hallmark of age-associated tissue dysfunction and cancer. Studies using mouse and fruit fly models have greatly improved our understanding of the functional contribution of the Wnt signalling pathway in adult intestinal biology. Here, we summarize the latest knowledge acquired from mouse and Drosophila research regarding canonical Wnt signalling and its key functions during stem cell driven intestinal homeostasis, regeneration, ageing and cancer.